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Haemodynamic consequences of acute pulmonary embolism predict risk of CTPA‐related acute kidney injury
Introduction Acute pulmonary embolism (APE) is the most severe clinical manifestation of venous thromboembolism (VTE).1, 2 According to the literature, chronic kidney disease (CKD) is related to higher risk of VTE as compared with subjects without renal disorders.3 Studies show that approximately 36% of patients with APE have impaired renal function, defined as an estimated glomerular filtration rate (eGFR) below 60 mL/min/1.73 m2 upon admission.
Plasma Troponins Identify Patients with Very Low-Risk Acute Pulmonary Embolism
Open AccessArticle by Bartosz Karolak 1,*, Michał Ciurzyński 1, Marta Skowrońska 1, Katarzyna Kurnicka 1, Magdalena Pływaczewska 2, Aleksandra Furdyna 1, Katarzyna Perzanowska-Brzeszkiewicz 1, Barbara Lichodziejewska 1, Szymon Pacho 1, Michał Machowski 1, Piotr Bienias 1, Małgorzata Wiśniewska 3, Marek Gołębiowski 3 and Piotr Pruszczyk 1 1 Department of Internal Medicine & Cardiology, Medical University of Warsaw, Lindleya 4 St., 02-005 Warsaw, Poland 2 Department of Pulmonary Circulation,...
Heart Rate Variability Impairment Is Associated with Right Ventricular Overload and Early Mortality Risk in Patients with Acute Pulmonary Embolism
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