Beiying Deng
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Frontiers | Shared molecular features and potential diagnostic biomarkers between ulcerative colitis and sarcopenia: an integrative bioinformatics analysis with preliminary experimental validation
ORIGINAL RESEARCH article Front. Immunol., 15 April 2026 Sec.
UCP2 Upregulates ACSL3 to Enhance Lipid Droplet Release from Acinar Cells and Modulates the Sirt1/Smad3 Pathway to Promote Macrophage‐to‐Myofibroblast Transition in Chronic Pancreatitis
1 Introduction Chronic pancreatitis (CP) is a progressive, irreversible disease with a rising global incidence in recent years.[1] Its pathological features include acinar cell damage, immune cell infiltration, pancreatic atrophy, and interstitial fibrosis, which lead to both endocrine and exocrine pancreatic dysfunction.[2] Consequently, CP can result in pancreatic diabetes, pancreatic calcification, ductal stones, and an increased risk of pancreatic cancer.
Unveiling and validating the role of fatty acid metabolism in ulcerati
Back to Journals » Journal of Inflammation Research » Volume 17 Authors Deng B, Zhen J, Xiang Z, Li X , Tan C, Chen Y, He P, Ma J, Dong W Received 1 July 2024 Accepted for publication 28 August 2024 Published 13 September 2024 Volume 2024:17 Pages 6345—6362 DOI https://doi.org/10.2147/JIR.S479011 Checked for plagiarism Yes Review by Single anonymous peer review Peer reviewer comments 2 Editor who approved publication: Dr Tara Strutt Beiying Deng,1,2,* Junhai Zhen,3,* Zixuan...
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