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Benney T. Endoni

As seen in: MDPI, BioRxiv
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Articles

MIRO1 controls energy production and proliferation of vascular smooth muscle cells

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By Lan Qian, Olha M. Koval, Benney T. Endoni, Denise Juhr
| BioRxiv
Abstract Background: The outer mitochondrial Rho GTPase 1, MIRO1, mediates mitochondrial motility within cells, but implications for vascular smooth muscle cell (VSMC) physiology and its roles in vascular diseases, such as neointima formation following vascular injury are widely unknown. Methods: Carotid ligation was performed in an in vivo model of selective Miro1 deletion in smooth muscle cells.
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MIRO1 Is Required for Dynamic Increases in Mitochondria-ER contact sites and Mitochondrial ATP During the Cell Cycle

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By Benney T. Endoni, Olha M. Koval, Chantal Allamargot, Tara Kortlever
| MDPI
All articles published by MDPI are made immediately available worldwide under an open access license. No special permission is required to reuse all or part of the article published by MDPI, including figures and tables. For articles published under an open access Creative Common CC BY license, any part of the article may be reused without permission provided that the original article is clearly cited. For more information, please refer to https://www.mdpi.com/openaccess.
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MIRO1 controls energy production and proliferation of smooth muscle cells

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By Lan Qian, Olha M. Koval, Denise Juhr, Benney T. Endoni
| BioRxiv
Abstract Background: The outer mitochondrial Rho GTPase 1, MIRO1, mediates mitochondrial distribution within cells and facilitates communication between mitochondria and the rest of the cell, but its roles in cardiovascular diseases, such as neointima formation following vascular injury are widely unknown. Methods: An in vivo model of selective Miro1 deletion in vascular smooth muscle cells (VSMCs) was generated, and the animals were subjected to carotid artery ligation.
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