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Articles by Bing Luan
GUSB-enriched sympathetic neurons promote thermogenic recruitment through extracellular matrix remodelling in male mice
Abstract Sympathetic neurons stimulate adipose thermogenic recruitment, which is further sustained by extracellular matrix (ECM) remodelling; however, the direct link between sympathetic neurons and ECM remodelling is still missing. Here we show the presence of a subpopulation of β-glucuronidase (GUSB)-enriched sympathetic neurons that drive ECM reorganization essential for thermogenic programming in male mice.
TH-positive pulmonary nerve fiber-epithelial interaction promotes adipose thermogenesis in male mice
Abstract Cold exposure activates sympathetic neurons in adipose tissue to maintain thermal homeostasis. However, whether sympathetic activation in non-adipose tissues contributes to cross-organ regulation of adipose thermogenesis remains unclear. Here, we demonstrate the activation of TH-positive pulmonary nerve fibers (TPNFs) by cold exposure, which further promotes adipose thermogenesis.
Pulmonary cold sensing through Club cells triggers adipose thermogenesis to ameliorate obesity
Keywords pulmonary cold sensing KCNK2 Club cell adipose thermogenesis obesity Get full text access Log in, subscribe or purchase for full access. References 1. Gregg, E.W. ∙ Shaw, J.E. Global Health Effects of Overweight and Obesity N. Engl. J. Med. 2017; 377:80-81 2. Betz, M.J. ∙ Enerbäck, S. Targeting thermogenesis in brown fat and muscle to treat obesity and metabolic disease Nat. Rev. Endocrinol. 2018; 14:77-87 3. Cohen, P. ∙ Kajimura, S. The cellular and functional complexity of thermogenic fat Nat. Rev.
Targeting parvalbumin promotes M2 macrophage polarization and energy expenditure in mice - Nature Communications
Abstract Exercise benefits M2 macrophage polarization, energy homeostasis and protects against obesity partially through exercise-induced circulating factors. Here, by unbiased quantitative proteomics on serum samples from sedentary and exercised mice, we identify parvalbumin as a circulating factor suppressed by exercise. Parvalbumin functions as a non-competitive CSF1R antagonist to inhibit M2 macrophage activation and energy expenditure in adipose tissue.
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