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Bjørnar Hassel

As seen in: Wiley Online Library, MDPI, Journal of Medical Case Reports, Medscape, The Journal of Clinical Investigation
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Articles by Bjørnar Hassel

Assessment of Developmental Prosopagnosia in an Individual with Tourette Syndrome and Attention Deficit Hyperactivity Disorder: A Case Report

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By Petter Espeseth Emhjellen, Randi Starrfelt, Rune Raudeberg, Bjørnar Hassel
| MDPI
All articles published by MDPI are made immediately available worldwide under an open access license. No special permission is required to reuse all or part of the article published by MDPI, including figures and tables. For articles published under an open access Creative Common CC BY license, any part of the article may be reused without permission provided that the original article is clearly cited. For more information, please refer to https://www.mdpi.com/openaccess.
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Glyceraldehyde metabolism in mouse brain and the entry of blood‐borne glyceraldehyde into the brain

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By Bjørnar Hassel, Ahmed Elsais, Patricia Cordero, Kristian Sørnes
| Wiley Online Library Verified
AGE advanced glycation end products GABA γ-aminobutyric acid i.v. intravenous NMRS nuclear magnetic resonance spectroscopy TCA cycle tricarboxylic acid cycle TMSP trimethylsilylpropionate 1 INTRODUCTION D-Glyceraldehyde (hereafter “glyceraldehyde”) is a reactive metabolite of several precursors, among them glucose, fructose, glycerol, and glycerate (Hassel et al., 2015; Hoffman et al., 1980; Leicht et al., 1978; O'Brien & Schofield, 1980; Stewart et al., 1967).
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Improvement of ADHD in Three Men During Testosterone Treatment

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By Ane Gretesdatter Rogne, Bjørnar Hassel
| Medscape Verified
Authors and Disclosures Ane Rogne1 and Bjørnar Hassel1,2* 1Department of Neurohabilitation, Oslo University Hospital, Kirkeveien 166, 0450 Oslo, Norway. 2Institute of Clinical Medicine, University of Oslo, Oslo, Norway. *Correspondence bjornar.hassel@medisin.uio.no Author contributions AR was responsible for CPT3 testing; BH was responsible for neurological examination, testosterone treatment, and medical follow-up. AR and BH wrote the manuscript. Both authors read and approved the final manuscript.
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Neuronal Pyruvate Carboxylation Supports Formation of Transmitter Glutamate

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By Bjørnar Hassel, Anders Bråthe
| JNeurosci Verified
Abstract Release of transmitter glutamate implies a drain of α-ketoglutarate from neurons, because glutamate, which is formed from α-ketoglutarate, is taken up by astrocytes. It is generally believed that this drain is compensated by uptake of glutamine from astrocytes, because neurons are considered incapable of de novosynthesis of tricarboxylic acid cycle intermediates, which requires pyruvate carboxylation.
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