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Breanne M Barrow on Muck Rack

Breanne M Barrow

As seen in: Diabetes Magazine, Journal of Endocrinology
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Articles

Going viral in the islet: mediators of SARS-CoV-2 entry beyond ACE2

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By Pandora L. Wander, Breanne M Barrow, Sakeneh Zraika, Rohita Rangu
Abstract Coronavirus disease 2019 (COVID-19) is caused by severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) infection. Following initial infection of airway epithelia, SARS-CoV-2 invades a wide range of cells in multiple organs, including pancreatic islet cells. Diabetes is now recognised as a risk factor for severe COVID-19 outcomes, including hospitalisation and death. Additionally, COVID-19 is associated with a higher risk of new-onset diabetes and metabolic complications of diabetes.
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Neprilysin Is Required for Angiotensin-(1–7)’s Ability to Enhance Insulin Secretion via Its Proteolytic Activity to Generate Angiotensin-(1–2)

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By Breanne M Barrow, Matthew Watson, Gurkirat S. Brar, Ryan Griesbach
| Diabetes Magazine Verified
Neprilysin Is Required for Angiotensin-(1–7)’s Ability to Enhance Insulin Secretion via Its Proteolytic Activity to Generate Angiotensin-(1–2) Gurkirat S. Brar, Breanne M. Barrow, Matthew Watson, Ryan Griesbach, Edwina Choung, Andrew Welch, Bela Ruzsicska, Daniel P. Raleigh, Sakeneh Zraika Diabetes Aug 2017, 66 (8) 2201-2212; DOI: 10.2337/db16-1318
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High fat feeding unmasks variable insulin responses in male C57BL/6 mouse substrains

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By Rebecca Hull Verified, Joshua R Willard, Matthias D Struck, Breanne M Barrow
| Journal of Endocrinology Verified
Mouse models are widely used for elucidating mechanisms underlying type 2 diabetes. Genetic background profoundly affects metabolic phenotype; therefore selecting the appropriate model is critical. While variability in metabolic responses between mouse strains is now well-recognized, it also occurs within C57BL/6 mice, of which several substrains exist. This within-strain variability is poorly understood, and could emanate from genetic and/or environmental differences.
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