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Glia-neuron interactions underlie a number of homeostatic processes in the brain. In this issue of Cell Metabolism, Li et al. (2020) Li F. Sami A. Noristani H.N. Slattery K. Qiu J. Groves T. Wang S. Veerasammy K. Chen Y.X. Morales J. et al. Glial metabolic rewiring promotes axon regeneration and functional recovery in the central nervous system. Cell Metab.
Highlights • Mice with 500 C9ORF72 repeats expressed repeat-containing RNA and dipeptide proteins • In two distinct labs, no survival or motor deficits were found linked to the transgene • Neurological phenotypes inherent to FVB mice complicate their use in ALS/FTD research Summary , published in A hexanucleotide repeat expansion at C9ORF72 is the most common genetic cause of amyotrophic lateral sclerosis (ALS)/frontotemporal dementia (FTD).
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