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Articles

Non-additive effects of schizophrenia risk genes reflect convergent downstream function

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By PJ Michael Deans, Jessica Johnson, Judit González, Carin Seah
| medRxiv
Abstract Genetic studies of schizophrenia (SCZ) reveal a complex polygenic risk architecture comprised of hundreds of risk variants, the majority of which are common in the population at-large and confer only modest increases in disorder risk. Precisely how genetic variants with individually small predicted effects on gene expression combine to yield substantial clinical impacts in aggregate is unclear.
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