Skip to main content
Chaoying Wei on Muck Rack

Chaoying Wei

As seen in: Color Research & Application, Wiley Online Library
Is this you? As a journalist, you can create a free Muck Rack account to customize your profile, list your contact preferences, and upload a portfolio of your best work. Claim your profile

Get in touch with Chaoying

Contact Chaoying, search articles and posts on X, monitor coverage, and track replies from one place.

Learn more about Muck Rack

Actions

Share this page

Is this you?

As a journalist, you can create a free Muck Rack account to customize your profile, list your contact preferences, and upload a portfolio of your best work.
Claim your profile

Articles

Targeting PKC as a Therapeutic Strategy to Overcome Chemoresistance in TNBC by Restoring Aurora Kinase B Expression

|
By Jinxin Chen, Guojie Long, Chaoying Wei, Vera Katalina
| Wiley Online Library Verified
1 Introduction Breast cancer is the most common cancer and the second leading cause of death among women worldwide [1]. It is considered a heterogeneous disease with substantial morphological, pathological and genetic variation. It can be classified into four major subtypes depending on the expression of hormone receptors (progesterone receptor and oestrogen receptor) and human epidermal growth factor receptor (HER2).
Open in Who Shared Issue with article?

Hyper activation of β-catenin signalling induced by IKKε inhibition thwarts colorectal cancer cell proliferation

|
By Jie Chen, Jun Zhao, Xuan Chen, Katie Lee, Zanxian Xia, Jian Peng, Chengming Ding, Zeming Jia, Yaoting Zhang, Yuzheng Zhou, Chaoying Wei, Jiantai He
| Color Research & Application Verified
Aberrant activation of Wnt/?-catenin signalling contributes significantly to the development of human colorectal cancers and ?-catenin is the key signalling molecule transducing canonical Wnt/?-catenin signalling. Therefore, ?-catenin is a promising therapeutic target for cancer treatment. This study demonstrates that the oncogenic IKK? kinase phosphorylates ?-catenin to restrain its hyper activation, therefore promoting colorectal cancer (CRC) cell proliferation. IKK?
Open in Who Shared Issue with article?
See all 2 articles