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Abstract Doxorubicin (DOX)-induced ovarian toxicity is a major cause of chemotherapy-related infertility in young female cancer patients, yet the underlying inflammatory mechanisms remain incompletely understood. In this study, we investigated whether CCL8-associated inflammatory remodeling contributes to DOX-induced ovarian injury and evaluated the therapeutic potential of localized chemokine inhibition using Bindarit. A murine model of DOX-induced ovarian injury was established.
As a journalist, you can create a free Muck Rack account to customize your profile, list your contact preferences, and upload a portfolio of your best work.
