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Cheung Kwan Yeung

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Articles

TP53 Loss Elevates NF-κB-IFN-β-MHC-Ia Signaling to Promote NK Cell Resistance in Osteosarcoma

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By Guihui Qin, Ye Yi, Cheung Kwan Yeung
| Wiley Online Library Verified
1 Introduction Osteosarcoma (OS) is the most common bone cancer originating from mesenchymal cells. It is highly heterogeneous, exhibiting remarkable variations in genetic makeup, behavior, and response to treatment [1]. The tumor suppressor gene TP53 is one of the most frequently mutated genes in diverse cancers, resulting in various statuses including wild type (WT), gain-of-function (GOF), dominant-negative (DN) effect, and loss-of-function (LOF) mutations [2, 3].
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