Skip to main content
Clint Miller on Muck Rack

Clint Miller

As seen in: Nature, BMJ Open, BMJ Open Sport & Exercise Medicine, MDPI, BioRxiv, BiggerPockets, medRxiv, Outdoor News, Green Living Magazine, Kalona News and

Get in touch with Clint

Contact Clint, search articles and posts on X, monitor coverage, and track replies from one place.

Learn more about Muck Rack

Actions

Share this page

Clint Miller’s Biography

Read Full Bio →

This grammarly app App is specially designed for Checking English Grammar, if you have any problem in understanding English grammar then install our app.

Articles

ERG preserves endothelial identity to limit atherosclerosis

|
By Steven R. Botts, Corey A. Scipione, Kristen Schulz, Kai Ellis, Christy Ho, Sneha Raju, Kamalben Prajapati, Kai Yu, Aniqa B. Khan, Chanele K. Polenz, Sharon J. Hyduk, Christian Cao, Joshua D. Wythe, Clint Miller, Myron I. Cybulsky, Jason E. Fish, Kathryn Howe, Leandro C. D. Breda, Stylianos Z. Prattas
| Nature Verified
Abstract Endothelial function is a safeguard against atherosclerosis, yet endothelial‑to‑mesenchymal transition (EndMT) can occur at atheroprone sites. How endothelial identity loss contributes to atherogenesis remains unclear. Here we investigated the role of the ETS transcription factor ERG, a key regulator of endothelial identity, in atheroprogression. Inducible endothelial Erg deletion increased plaque burden in hypercholesterolemic mice.
Open in Who Shared Issue with article?

High LTV Bridge Lenders

|
By Givo Aghasi, Daniel Segovia, Clint Miller
| BiggerPockets Verified
I have a 12 unit property in Lakewood I am in contract on. I'm looking at doing a bridge while I raise rents to market (all leases MTM) Any reccomendations ? Shooting for the holy grail of 20% down with minimal prepay
Open in Who Shared Issue with article?

Interferon dependent immune memory during HSV-1 neuronal latency via increased H3K9me3 and restriction by ATRX - Nature Communications

|
By Gaelle Auguste, Alison K Francois, David J. Picketts, Chris Boutell, Clint Miller, Sarah E. Dremel
| Nature Verified
Abstract Herpes simplex virus-1 (HSV-1) establishes a latent infection in neurons, periodically reactivating to cause disease. Neuronal conditions, including immune signaling, during initial HSV-1 infection, impact later reactivation. Type I interferon (IFNα) exposure during initial infection results in promyelocytic leukemia nuclear-body (PML-NB) formation and subsequent restriction of reactivation, via mechanisms that were unknown.
Open in Who Shared Issue with article?
See all 20 articles