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Davide Fraticelli on Muck Rack

Davide Fraticelli

As seen in: Nature, Neurology
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Articles

Structural and molecular basis of specialized translation mediated by the ribosome mRNA-binding channel

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By Davide Fraticelli, Benjamin Weiss, Ariel Ogran, Anat Bashan, Ada Yonath, Disha Gajanan Hiregange, Tal Havkin-Solomon, Irene Martinez Roman, Rivka Dikstein
| Nature Verified
Abstract The ribosome mRNA channel is central to translation, yet its role in regulatory mechanisms remains unclear. Using cryo-EM of human ribosomal complexes bound to Kozak and TISU mRNAs from wild-type (WT) and RPS26/eS26 mutant (RPS26dC) cells, we demonstrate that both RPS26/eS26 and mRNA adopt distinct conformations, explaining the opposing effects of RPS26dC on their activity.
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Assessment of Sacsin Turnover in Patients With ARSACS

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By Fabiana Longo, Daniele De Ritis, Annarita Miluzio, Davide Fraticelli
| Neurology Verified
Abstract Background and Objectives Autosomal recessive spastic ataxia of Charlevoix-Saguenay (ARSACS) is caused by variations in SACS gene encoding sacsin, a huge multimodular protein of unknown function. More than 200 SACS variations have been described worldwide to date.
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Assessment of Sacsin Turnover in Patients With ARSACS: Implications for Molecular Diagnosis and Pathogenesis

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By Annarita Miluzio, Fabiana Longo, Daniele De Ritis, Davide Fraticelli
| Neurology Verified
October 14, 2021Research ArticleOpen Access Fabiana Longo, Daniele De Ritis, Annarita Miluzio, Davide Fraticelli, Jonathan Baets, Marina Scarlato, Filippo M. Santorelli, Stefano Biffo, Francesca Maltecca First published October 14, 2021, DOI: https://doi.org/10.1212/WNL.0000000000012962 Abstract Background and Objectives: Autosomal recessive spastic ataxia of Charlevoix-Saguenay (ARSACS) is caused by mutations in SACS gene encoding sacsin, a huge multimodular protein of unknown function.
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