Denisa Bianca Mercean on Muck Rack

Denisa Bianca Mercean

As seen in: Nature, MDPI
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Articles

Circulating ACE2 levels and ACE I/D polymorphism in severe aortic stenosis

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By Denisa Bianca Mercean, Ioana Berindan-Neagoe, Dana Pop, Adela Serban, Carmen Stanca Melincovici, Carmen Mihaela Mihu
| Nature Verified
Abstract The renin–angiotensin system (RAS) contributes to the fibro-calcific remodeling characteristic of aortic stenosis (AS). Although increased circulating ACE2 enzymatic activity has been reported in severe AS, data on circulating ACE2 protein concentration, ACE levels, and their relationship with ACE genetic variation remain limited.
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JPM | Free Full-Text | The Impact of Monocyte to High-Density Lipoprotein Cholesterol Ratio on All-Cause and Cardiovascular Mortality in Patients with Transcatheter Aortic Valve Replacement

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By Raluca Tomoaia, Adela Serban, Denisa Bianca Mercean, Radu Hagiu
| MDPI
1. Introduction Calcific aortic stenosis (AS) is the most common heart valve disorder in developed countries [1]. It is identified by the progressive remodeling and thickening of the aortic valve leaflets due to fibro-calcific changes. For a long time, calcific AS was considered to be a passive process of degeneration due to mechanical stress over the years.
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