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Articles by Dinglan Wu
GITR activation potentiates anti-tumor immunity of tumor-infiltrating lymphocytes expanded from glioblastoma by rescuing exhaustion - Oncogene
Abstract Autologous tumor-infiltrating lymphocyte (TIL) therapy holds transformative potential for solid tumors, yet its efficacy in glioblastoma remains limited by T cell exhaustion and immunosuppression. In the current study, we optimized an effective and reliable method for in vitro expansion of TILs from glioblastoma lesions and assessed their tumor-killing capacity both in vitro and in vivo.
UMP functions as an endogenous regulator of NR4A1 to control gastric cancer progression
Keywords NR4A1 UMP gastric cancer de novo pyrimidine biosynthesis pathway DHODH Get full text access Log in, subscribe or purchase for full access. References 1. Bray, F. ∙ Laversanne, M. ∙ Sung, H. ... Global cancer statistics 2022: GLOBOCAN estimates of incidence and mortality worldwide for 36 cancers in 185 countries CA Cancer J. Clin. 2024; 74:229-263 2. Wang, R. ∙ Dang, M. ∙ Harada, K. ...
UMP functions as an endogenous regulator of NR4A1 to control gastric cancer progression
Keywords NR4A1 UMP gastric cancer de novo pyrimidine biosynthesis pathway DHODH Get full text access Log in, subscribe or purchase for full access. References 1. Bray, F. ∙ Laversanne, M. ∙ Sung, H. ... Global cancer statistics 2022: GLOBOCAN estimates of incidence and mortality worldwide for 36 cancers in 185 countries CA Cancer J. Clin. 2024; 74:229-263 2. Wang, R. ∙ Dang, M. ∙ Harada, K. ...
Nuclear receptor TLX functions to promote cancer stemness and EMT in prostate cancer via its direct transactivation of CD44 and stem cell-regulatory transcription factors | British Journal of Cancer
Abstract Prostate cancer stem cells (PCSCs) play crucial roles in therapy-resistance and metastasis in castration-resistant prostate cancer (CRPC). Certain functional link between cancer stemness and epithelial-mesenchymal transition (EMT) is involved in CRPC. However, up-stream regulators controlling these two processes in PCSCs are still poorly understood.
Interferon-induced transmembrane protein-1 competitively blocks Ephrin receptor A2-mediated Epstein–Barr virus entry into epithelial cells - Nature Microbiology
Abstract Epstein–Barr virus (EBV) can infect both B cells and epithelial cells (ECs), causing diseases such as mononucleosis and cancer. It enters ECs via Ephrin receptor A2 (EphA2). The function of interferon-induced transmembrane protein-1 (IFITM1) in EBV infection of ECs remains elusive.
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