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Ela Bar

As seen in: Wiley Online Library, MDPI
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Articles

Neuronal deletion of Gtf2i results in developmental microglial alterations in a mouse model related to Williams syndrome

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By Ela Bar, Inbar Fischer, May Rokach, Galit Elad-Sfadia
| Wiley Online Library Verified
1 INTRODUCTION Williams syndrome (Kozel et al., 2021; Nir & Barak, 2021) (WS) is a genetic neurodevelopmental disorder caused by a heterozygous micro-deletion of ~26–28 genes from the WS critical region and characterized by hyper-sociability and unique neurocognitive abnormalities (Barak & Feng, 2016). Of the deleted genes, GTF2I has been linked to hyper-sociability in WS (Chailangkarn et al., 2018; Ophir et al., 2023; Procyshyn et al., 2017; Sakurai et al., 2011).
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Deletion of Gtf2i via Systemic Administration of AAV-PHP.eB Virus Increases Social Behavior in a Mouse Model of a Neurodevelopmental Disorder

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By Gilad Levy, Ela Bar, Omer Ophir, Omri Kimchi Feldhorn
| MDPI
Altmetric share Share announcement Help format_quote Cite thumb_up ... Endorse textsms ... Comment Need Help? Support Find support for a specific problem in the support section of our website. Get Support Feedback Please let us know what you think of our products and services. Give Feedback Information Visit our dedicated information section to learn more about MDPI.
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Hyperbaric Oxygen Therapy Alleviates Social Behavior Dysfunction and Neuroinflammation in a Mouse Model for Autism Spectrum Disorders

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By Inbar Fischer, Sophie Shohat, Gilad Levy, Ela Bar
| MDPI
Open AccessArticle by Inbar Fischer 1, Sophie Shohat 2, Gilad Levy 1, Ela Bar 3,4, Sari Schokoroy Trangle 3, Shai Efrati 1,5 and Boaz Barak 1,3,* 1 The Sagol School of Neuroscience, Tel Aviv University, Tel Aviv 69978, Israel 2 Department of Biomedical Engineering, Tel Aviv University, Tel Aviv 69978, Israel 3 The School of Psychological Sciences, Tel Aviv University, Tel Aviv 69978, Israel 4 The School of Neurobiology, Biochemistry & Biophysics, Faculty of Life Sciences, Tel Aviv University,...
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