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Keywords senescence ferroptosis PUFA DGLA aging senolytic metabolism lipid peroxidation obesity desaturation Introduction Throughout their lifespans, cells are subject to various stressors, including macromolecular damage, epigenetic disruption, metabolic shifts, and mitochondrial dysfunction.1,2 If unresolved, accumulation of these insults over time can result in degenerative conditions and cancer, necessitating activation of protective cellular responses.
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