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Translational inhibition of APP by Posiphen: Efficacy, pharmacodynamics, and pharmacokinetics in the APP/PS1 mouse

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By Andrew F. Teich, Ekta Sharma, Eliza Barnwell, Hong Zhi Zhang
| trci.alzdem.com
Alzheimer's disease (AD) is the leading cause of dementia in the elderly. The hallmarks of AD at the microscopic level are senile plaques and neurofibrillary tangles (NFTs), formed by aggregation of amyloid ? (A?) peptide, mostly of 42-aa length, and hyperphosphorylated tau protein, respectively [1][1]. The A?
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