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Fibroblast Has2 limits acute heart failure following myocardial infarction in male mice

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By Danielle Little, Caitlin Howard, Emma Pendergraft, Kenneth R. Brittian
| Wiley Online Library Verified
1 INTRODUCTION Myocardial infarction (MI) activates cardiac fibroblasts, which participate in acute scar formation and remodeling (Frangogiannis, 2016). The function of cardiac fibroblasts in such replacement fibrosis is essential, and limitations to their function could impair the acute wound healing response in the heart (Daseke et al., 2020).
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