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Enes K. Ergin

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Articles

ProteoForge: An Imputation-Aware Framework for Differential Proteoform Discovery in Bottom-Up Proteomics Click to copy article link Article link copied!

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By Enes K. Ergin, Kirsty Ferguson, Agustina Conrrero
| dx.doi.org
ArticleJune 9, 2026 * Enes K. Ergin Department of Pathology and Laboratory Medicine, University of British Columbia, Vancouver, British Columbia V6T 1M9, Canada Michael Cuccione Childhood Cancer Research Program, BC Children’s Hospital Research Institute, Vancouver, British Columbia V5Z 4H4, Canada *Email: eergin@bcchr.ca Agustina Conrrero Michael Cuccione Childhood Cancer Research Program, BC Children’s Hospital Research Institute, Vancouver, British Columbia V5Z 4H4, Canada Kirsty M.
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Statistical Testing for Protein Equivalence Identifies Core Functional Modules Conserved across 360 Cancer Cell Lines and Presents a General Approach to Investigating Biological Systems

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By Enes K. Ergin, Philipp F. Lange, Junia J.K. Myung
| dx.doi.org
Quantitative proteomics has enhanced our capability to study protein dynamics and their involvement in disease using various techniques, including statistical testing, to discern the significant differences between conditions. While most focus is on what is different between conditions, exploring similarities can provide valuable insights. However, exploring similarities directly from the analyte level, such as proteins, genes, or metabolites, is not a standard practice and is not widely adopted.
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Targetable lesions and proteomes predict therapy sensitivity through disease evolution in pediatric acute lymphoblastic leukemia - Nature Communications

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By C. Lim, Christopher Maxwell, Enes K. Ergin, Nina Rolf
| Nature Verified
Abstract Childhood acute lymphoblastic leukemia (ALL) genomes show that relapses often arise from subclonal outgrowths. However, the impact of clonal evolution on the actionable proteome and response to targeted therapy is not known. Here, we present a comprehensive retrospective analysis of paired ALL diagnosis and relapsed specimen. Targeted next generation sequencing and proteome analysis indicate persistence of actionable genome variants and stable proteomes through disease progression.
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