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Esam Khanfar

As seen in: Frontiers in Immunology, MDPI
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Articles

TAPBPR promotes editing of the HLA-B44 peptide repertoire, increasing the presentation of peptides containing a C-terminal tryptophan

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By Aure Aflalo, Marcel Wacker, Esam Khanfar, Jens Bauer, Juliane S. Walz, Louise Boyle Verified, Arwen F. Altenburg
| Frontiers in Immunology
1 Introduction Major histocompatibility complex class I (MHC-I) molecules play a crucial role in the adaptive immune response by presenting antigenic peptides typically derived from the intracellular proteome to CD8+ T cells (1, 2). Furthermore, the expression of MHC-I is monitored by natural killer (NK) cells for immunosurveillance (3, 4).
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The Spleen Modulates the Balance of Natural and Pathological Autoantibodies in a Mouse Model of Autoimmune Arthritis

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By Katalin Olasz, Szonja Gál, Esam Khanfar, Peter Balogh
| MDPI
All articles published by MDPI are made immediately available worldwide under an open access license. No special permission is required to reuse all or part of the article published by MDPI, including figures and tables. For articles published under an open access Creative Common CC BY license, any part of the article may be reused without permission provided that the original article is clearly cited. For more information, please refer to https://www.mdpi.com/openaccess.
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Natural and Pathological Autoantibodies Show Age-Related Changes in a Spontaneous Autoimmune Mouse (NZB) Model

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By Szonja Gál, Erzsébet Gajdócsi, Esam Khanfar, Katalin Olasz
| MDPI
Abstract : The natural autoantibody (natAAb) network is thought to play a role in immune regulation. These IgM antibodies react with evolutionary conserved antigens; however, they do not lead to pathological tissue destruction as opposed to pathological autoantibodies (pathAAb).
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