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Articles by Eva Bartok
DHX36 is a regulatory switch in the interferon-mediated antiviral response
Abstract INTRODUCTION RESULTS DISCUSSION MATERIALS AND METHODS Acknowledgments Supplementary Materials REFERENCES Information & Authors Metrics & Citations View Options References Figures Tables Media Share Abstract Innate immune and stress responses must be tightly regulated to prevent aberrant activation in the absence of pathogens. The RNA helicase DHX36 has been implicated in viral RNA sensing, but its role in immune regulation is not fully understood.
Blood and Black Lace 1964 1080p BluRay x264-OFT
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Blutige Seide am 13. Dezember 2025 um 09:05 Uhr auf Sky Cinema Classics HD
Heute 09:05 - 10:40 95 min. Sky Cinema Classics HD HD16:9 D 1964 Thriller Stilprägender italienischer Thrillerklassiker von Mario Bava: Mehrere Models einer Agentur werden ermordet. Da taucht das Tagebuch eines der Opfer auf - mit brisanten Infos.
Come together, right now! ZCCHC3 orchestrates cytosolic nucleic acid sensing through phase condensation
Get full text access Log in, subscribe or purchase for full access. References 1. Shi, M. ∙ Jiang, T. ∙ Zhang, M. ... Nucleic acid-induced ZCCHC3 condensation promotes broad innate immune responses Mol. Cell. 2025; 85 2. Aceituno-Valenzuela, U. ∙ Micol-Ponce, R. ∙ Ponce, M.R. Genome-wide analysis of CCHC-type zinc finger (ZCCHC) proteins in yeast, Arabidopsis, and humans Cell. Mol. Life Sci. 2020; 77:3991-4014 3. Lian, H. ∙ Zang, R. ∙ Wei, J. ...
Chronic endoplasmic reticulum stress in myotonic dystrophy type 2 promotes autoimmunity via mitochondrial DNA release - Nature Communications
Abstract Myotonic dystrophy type 2 (DM2) is a tetranucleotide CCTG repeat expansion disease associated with an increased prevalence of autoimmunity. Here, we identified an elevated type I interferon (IFN) signature in peripheral blood mononuclear cells and primary fibroblasts of DM2 patients as a trigger of chronic immune stimulation.
Unexpected bonds: Ubiquitin-like conjugation of cGAS/CD-NTases supports their enzymatic activity and antiphage defense - Signal Transduction and Targeted Therapy
In their recently published study in Nature, Jenson et al.1 provide compelling, unexpected evidence that bacterial cGAMP synthase (cGAS) and other cGAS/DncV-like Nucleotidyltransferases (CD-NTase) undergo ubiquitin-like conjugation (ULC) in type-II cyclic-oligonucleotide-based anti-phage signaling systems (CBASS). cGAS conjugation promotes cyclic-nucleotide (CN) formation and thus antiphage defense.
Influenza A Infection Stimulates RIG-I and Enhances Effector Function of Primary Human NK Cells
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ADAR1 edits the SenZ and SenZ-ability of RNA
Some RNAs can assume a Z conformation, an unusual, left-handed turn. In this issue of Immunity, three studies report that mutations in the Zα-RNA binding domain of the adenosine deaminase ADAR1 are sufficient to induce autoinflammatory disease in mice, which models human Aicardí-Goutières syndrome, highlighting the important role of Z-RNA editing in limiting innate immune recognition of endogenous RNA.
Immune Sensing Mechanisms that Discriminate Self from Altered Self and Foreign Nucleic Acids
All lifeforms have developed highly sophisticated systems equipped to detect altered self and non-self nucleic acids (NA). In vertebrates, NA-sensing receptors safeguard the integrity of the organism by detecting pathogens, dyshomeostasis and damage, and inducing appropriate responses to eliminate pathogens and reconstitute homeostasis. Effector mechanisms include i) immune signaling, ii) restriction of NA functions such as inhibition of mRNA translation, and iii) cell death pathways.
BAX/BAK-Induced Apoptosis Results in Caspase-8-Dependent IL-1β Maturation in Macrophages
Antibodies BAX Cell Signaling Technology Cat#2772 BAK (Polyclonal rabbit) BD Biosciences Cat#556396 BCL2 (Purified mouse) Clone 7/Bcl-2 BD Biosciences Cat#610539 Beta actin antibody (C4) Santa Cruz Biotechnology Cat#Sc-47778 BID (Human/Mouse) R&D systems Cat#AF860 Caspase-1 (p20) (human), mAb (Bally-1) Adipogen International Cat#AG-20B-0048-C100 Caspase-1 (p20) (mouse), mAb (Casper-1) Adipogen International Cat#AG-20B-0042-C100 Caspase-3 Cell Signaling Technology Cat#9662 Caspase-8 (Asp 387)...
Cutting Edge: Reactive Oxygen Species Inhibitors Block Priming, but Not Activation, of the NLRP3 Inflammasome
AbstractA common denominator among the multiple damage-inducing agents that ultimately lead to activation of NLRP3 has not yet been identified. Recently, production of reactive oxygen species (ROS) has been suggested to act as a common event upstream of the NLRP3 inflammasome machinery. Because de novo translation of NLRP3 is an essential step in the activation of NLRP3, we investigated the role of substances that inhibit either ROS production or its oxidative activity.
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