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Eveljn Scarian

As seen in: Nature, MDPI
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Articles

Proteasomal-dependent CHK1 degradation leads to DNA damage accumulation in ALS cellular model systems

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By Stefania Modafferi, Valentina Silenzi, Anna Garbelli, Eveljn Scarian, Sara D’Uva, Mauro Cozzolino, Orietta Pansarasa, Nadia D’Ambrosi, Simone Sabbioneda, Mariangela Morlando, Sofia Francia, Gloria Lazoi, Tiziana Santini, Adelaide Riccardi
| Nature Verified
Abstract Amyotrophic lateral sclerosis (ALS) is characterised by the aggregation of TDP-43 and mutant FUS in the cytoplasm of affected motor neurons. Accumulation of DNA damage is emerging as a novel correlative trait of ALS. We recently showed that formation of TDP-43 and FUS cytoplasmic inclusions (CIs) lead to DNA damage accumulation through dysregulation of the DNA damage response (DDR).
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New Insights into Oxidative Stress and Inflammatory Response in Neurodegenerative Diseases

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By Eveljn Scarian, Camilla Viola, Francesca Dragoni, Rosalinda Di Gerlando
| MDPI
All articles published by MDPI are made immediately available worldwide under an open access license. No special permission is required to reuse all or part of the article published by MDPI, including figures and tables. For articles published under an open access Creative Common CC BY license, any part of the article may be reused without permission provided that the original article is clearly cited. For more information, please refer to https://www.mdpi.com/openaccess.
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