Ezra Baraban
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GLI1‐Altered Mesenchymal Tumor of the Duodenum With a Novel TNFAIP3::GLI1 Gene Fusion: Report of a Diagnostically Challenging Case Associated With Late Metastasis
Disclosure The authors have nothing to report. Conflicts of Interest The authors declare no conflicts of interest. Data Availability Statement The data that support the findings of this study are available on request from the corresponding author. The data are not publicly available due to privacy or ethical restrictions. References 1, “Gene of the Month: GLI-1,” Journal of Clinical Pathology 73 (2020): 228–230.
Dynamics of Tumor-informed Circulating Tumor DNA and Association with Clinical Responses to Enfortumab and Pembrolizumab in Patients with Locally Advanced or Metastatic Urothelial Carcinoma
Get full text access Log in, subscribe or purchase for full access. References 1. Powles, T. ∙ Valderrama, B.P. ∙ Gupta, S. ... Enfortumab vedotin and pembrolizumab in untreated advanced urothelial cancer N Engl J Med. 2024; 390:875-888 2. Powles, T. ∙ Assaf, Z.J. ∙ Davarpanah, N. ... ctDNA guiding adjuvant immunotherapy in urothelial carcinoma Nature. 2021; 595:432-437 3. Christensen, E. ∙ Birkenkamp-Demtröder, K. ∙ Sethi, H. ...
Lesion-based indicators predict long-term outcomes of pheochromocytoma and paraganglioma- SIZEPASS
Introduction Chromaffin and neural progenitor cells share many features (1) and cross-differentiate from the same progenitors (2). Tumors derived from chromaffin progenitor cells in the adrenal medulla (3), or from autonomic paraganglia of the sympathetic or parasympathetic nervous system, define the group of non-epithelial neuroendocrine tumors (4). However, nestin-positive progenitor cells or Sox-10 deplete-initiated chromaffin cells may escape physiological controls to form tumors (5).
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