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Fadime Aydın Köse on Muck Rack

Fadime Aydın Köse

As seen in: Wiley Online Library, MDPI
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Articles

Anti‐Inflammatory Effects of Spexin on Acetic Acid‑Induced Colitis in Rats via Modulating the NF‐κB/NLRP3 Inflammasome Pathway

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By Fadime Aydın Köse, Sevil Arabacı Tamer, Sevinç Yanar, Özcan Budak
| Wiley Online Library Verified
1 Introduction Inflammatory bowel diseases (IBDs), including ulcerative colitis (UC), represent chronic gastrointestinal disorders characterized by persistent inflammation, diarrhea, abdominal discomfort, rectal bleeding, and unintended weight loss [1, 2]. The development of UC is multifaceted, involving an intricate interplay between genetic predisposition and environmental influences that disrupt the immune system, culminating in digestive and intestinal inflammation [2, 3].
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Novel Triazolopyrimidinone Compounds as Inhibitors of Adipocyte Fatty Acid-Binding Protein (FABP4)

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By Merve Saylam, Fadime Aydın Köse, Gunes Coban, Huseyin Istanbullu
| Wiley Online Library Verified
Conflicts of Interest The authors declare no conflicts of interest. Supporting Information Filename Description cbdv202500205-sup-0001-SuppMat.pdf1 MB Supporting-Information References 1, , , et al., “Global Burden of 87 Risk Factors in 204 Countries and Territories, 1990–2019: A Systematic Analysis for the Global Burden of Disease Study 2019,” Lancet 396, no. 10258 (2020): 1223–1249.
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Peroxiredoxins and Hypoxia-Inducible Factor-1α in Duodenal Tissue: Emerging Factors in the Pathophysiology of Pediatric Celiac Disease Patients

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By Sema Aydogdu, Fadime Aydın Köse, Aysun Pabuccuoglu, Miray Karakoyun
| MDPI
4.4. Peroxiredoxins mRNA and Protein Expressions in Duodenal Tissue of Pediatric CD Patients The obtained RT-PCR and immunoblot data revealed for the first time that six peroxiredoxin isoforms were expressed in human duodenal tissue. Furthermore, it has been shown that PRDX 1, PRDX 2, PRDX 4 and PRDX 5 isoforms significantly altered in pediatric celiac patients compared to the control group. These findings support our hypothesis that PRDXs may be involved in the pathogenesis of CD.
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