Fernando Javier Barreyro

Background Functional dyspepsia (FD) is a multifactorial disorder. Helicobacter pylori (H. pylori)-related dyspepsia (HpD) may be considered a separate entity. Duodenal eosinophilia is a potential pathogenic mechanism in FD. However, the impact of duodenal eosinophilia and host genetic polymorphism of innate and pro-inflammatory cascade, nucleotide-binding oligomerization domain 1 (NOD-1), and interleukin-1 beta (IL-1β) in HpD was not explored.

INVITED EDITORIAL Juan Antonio Sorda, Corresponding Author Juan Antonio Sorda juansorda@gmail.com Department of Gastroenterology and Hepatology, University Hospital “José de San Martín”, Faculty of Medicine, University of Buenos Aires, Buenos Aires, Argentina Search for more papers by this author Esteban González Ballerga, Esteban González Ballerga Department of Gastroenterology and Hepatology, University Hospital “José de San Martín”, Faculty of Medicine, University of Buenos Aires, Buenos...