Skip to main content
Gali Epstein Shochet on Muck Rack

Gali Epstein Shochet

As seen in: Memory Studies, MDPI, European Respiratory Journal
Is this you? As a journalist, you can create a free Muck Rack account to customize your profile, list your contact preferences, and upload a portfolio of your best work. Claim your profile

Get in touch with Gali Epstein

Contact Gali Epstein, search articles and posts on X, monitor coverage, and track replies from one place.

Learn more about Muck Rack

Actions

Share this page

Is this you?

As a journalist, you can create a free Muck Rack account to customize your profile, list your contact preferences, and upload a portfolio of your best work.
Claim your profile

Articles

Prostaglandin E2 (PGE2) and Roflumilast Involvement in IPF Progression

|
By Noa Moshkovitz, Gali Epstein Shochet, David Shitrit, Epstein Shochet
| MDPI
3. Discussion The ECM plays a major role in IPF progression [15]. In this work, we utilized the already established IPF-CM system to explore the involvement of PGE2 in the IPF–HLF–ECM interplay.
Open in Who Shared Issue with article?

Galectin-3 levels are elevated following nintedanib treatment

|
By Gali Epstein Shochet, David Shitrit, Alon Pomerantz, Becky Bardenstein-Wald
| Memory Studies
Introduction Idiopathic pulmonary fibrosis (IPF) is considered the most common and severe form of pulmonary fibrosis, with a 5-year survival rate of only 20% reflecting the lack of effective therapies.1 The pathology of IPF is characterized by scar tissue formation (fibrosis) and excessive extracellular matrix deposition, resulting in the loss of lung function.
Open in Who Shared Issue with article?

LATE-BREAKING ABSTRACT: Fibroblast conditioned matrix: A new approach to study extracellular matrix (ECM) effects in idiopathic pulmonary fibrosis (IPF) | European Respiratory Society

|
By Sackler Faculty, Gali Epstein Shochet, Lilach Israeli-Shani
| European Respiratory Journal Verified
AbstractObjective: Idiopathic pulmonary fibrosis (IPF) is a progressive lung disease with poor prognosis. Activated fibroblasts are the key effector cells in fibrosis, producing excessive amounts of collagen and extracellular matrix (ECM) proteins. The microenvironment (e.g. ECM) plays a significant role in IPF progression.
Open in Who Shared Issue with article?
See all 3 articles