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Hanumakumar Bogireddi

As seen in: BioRxiv
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Articles

Pervasive nuclear envelope ruptures precede ECM signaling and disease onset without activating cGAS-STING in Lamin-cardiomyopathy mice

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By Atsuki En, Hanumakumar Bogireddi, Briana Thomas, Alexis Stutzman
| BioRxiv
Abstract Nuclear envelope (NE) ruptures are emerging observations in Lamin-related dilated cardiomyopathy, an adult-onset disease caused by loss-of-function mutations in Lamin A/C, a nuclear lamina component. Here, we tested a prevailing hypothesis that NE ruptures trigger pathological cGAS-STING cytosolic DNA-sensing pathway, using a mouse model of Lamin-cardiomyopathy.
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The cGAS-STING pathway is dispensable in a mouse model of LMNA-cardiomyopathy despite nuclear envelope rupture

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By Atsuki En, Hanumakumar Bogireddi, Briana Thomas, Alexis Stutzman
| BioRxiv
Abstract Mutations in the nuclear Lamin A/C gene (LMNA) cause diverse degenerative disorders, including malignant dilated cardiomyopathy in adults. A prevailing hypothesis postulates that LMNA mutations cause nuclear envelope ruptures that trigger pathogenic inflammatory signaling via the cGAS-STING cytosolic DNA-sensing pathway.
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YAP/TAZ are Crucial Regulator of Macrophage-mediated Pulmonary Inflammation and Fibrosis after Bleomycin-induced Injury

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By Manvendra Singh, Masum M. Mia, Dasan Mary Cibi, Hanumakumar Bogireddi
| BioRxiv
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