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Haofuzi Zhang

As seen in: Nature, MDPI
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Articles

High-altitude hypoxia aggravated neurological deficits in mice induced by traumatic brain injury via BACH1 mediating astrocytic ferroptosis - Cell Death Discovery

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By Haofuzi Zhang
| Nature Verified
Abstract Traumatic brain injury (TBI) is one of the leading causes of disability and mortality, which was classified as low-altitude TBI and high-altitude TBI. A large amount of literature shows that high-altitude TBI is associated with more severe neurological impairments and higher mortality rates compared to low-altitude TBI, due to the special environment of high-altitude hypoxia. However, the role of high-altitude hypoxia in the pathogenesis of TBI remains unclear.
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Preso enhances mGluR1-mediated excitotoxicity by modulating the phosphorylation of mGluR1-Homer1 complex and facilitating an ER stress after traumatic brain injury | Cell Death Discovery

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By Haofuzi Zhang
| Nature Verified
Abstract Glutamate receptor (GluR)-mediated excitotoxicity is an important mechanism causing delayed neuronal injury after traumatic brain injury (TBI).
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Role of Sirtuin 3 in Degenerative Diseases of the Central Nervous System

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By Haofuzi Zhang, Shuhui Dai, Yuefan Yang, Jialiang Wei
| MDPI
1. Introduction Neurodegenerative disorders are described by a progressive loss of neurons and by protein deposition, which are present in Alzheimer’s disease (AD), Parkinson’s disease (PD), Huntington’s disease (HD), amyotrophic lateral sclerosis (ALS), multiple sclerosis (MS), and so on [1,2]. As the population ages, neurodegenerative disorders put a significant burden on society and families while reducing the patients’ quality of life.
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