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Loading metrics Open Access Peer-reviewed Research Article ? This is an uncorrected proof. Citation: Subramaniam KV, Lim HJ, Wang B, Mihaylova VT, Thompson EN, Krause DS, et al. (2026) Dose-dependent activation of the Hippo pathway by Type I and Type III interferons suppresses tissue repair by human bronchial epithelial cells. PLoS Biol 24(1): e3003615. https://doi.org/10.1371/journal.pbio.3003615 Academic Editor: Michaela U.
Endoplasmic reticulum chaperones stabilize ligand-receptive MR1 molecules for efficient presentation of metabolite antigens Hamish E. G. McWilliam, Jeffrey Y. W. Mak, Wael Awad, Matthew Zorkau, Sebastian Cruz-Gomez, Hui Jing Lim, Yuting Yan, Sam Wormald, Laura F. Dagley, Sidonia B. G. Eckle, Alexandra J. Corbett, Haiyin Liu, Shihan Li, Scott J. J. Reddiex, Justine D. Mintern, Ligong Liu, James McCluskey, Jamie Rossjohn, David P. Fairlie, Jose A.
A human immune system sans MAITs Here, Howson et al. set out to determine the genetic basis of immunodeficiency in a patient with persistent tattoo-associated human papilloma virus–positive warts. Whole-exome sequencing led them to identify a rare point mutation Arg31His (R9H in the mature protein) in both copies of the MR1 gene. MR1 encodes an MHC-like molecule that presents bacteria-derived riboflavin metabolites to mucosal-associated invariant T (MAIT) cells.
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