Hui-Ying Liu

Abstract Background: Chlorine is a potent irritant gas with asphyxiant toxicity. Depending on the concentration and duration of exposure, intentional or accidental inhalation can rapidly cause marked injury to the respiratory tract and, in some cases, leave patients with sustained ventilatory impairment. At present, there is no approved antidote that specifically targets chlorine intoxication.

1 Introduction The pursuit of electronic systems with cognitive, decision-making, and interactive capabilities has driven significant advancements in artificial intelligence.[1-6] This endeavor has also catalyzed the development of advanced functional materials and devices optimized for information storage and processing.[7-15] A pivotal milestone in this field was the landmark study of Williams and Strukov in 2008, which spurred the rapid evolution of memristor technology.[16] Memristors,...

3. Discussion Our results demonstrated that the expression of VCAN was positively correlated with tumor stage, nodal metastasis status, histologic grade and histologic subtypes in the BLCA database. VCAN has previously been found to significantly increase in cases of lung inflammation [29,30,31]. VCAN regulates inflammation through the hyaluronic acid (HA) matrix [32,33]. Monocytes/macrophages and stromal fibroblasts are capable of secreting VCAN, which is involved in the formation of the HA matrix.