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Jaclyn A Kaiser on Muck Rack

Jaclyn A Kaiser

As seen in: Nature, PLOS, Future Medicine
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Articles

Intranasal respiratory syncytial virus vaccine attenuated by codon-pair deoptimization of seven open reading frames is genetically stable and elicits mucosal and systemic immunity and protection against challenge virus replication in hamsters

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By Megan Levy Verified, Jessica Chen, Jaclyn A Kaiser, Hong-Su Park, PLOS Pathogens
| PLOS
Loading metrics Open Access Peer-reviewed Research Article ? This is an uncorrected proof. Citation: Levy M, Chen JW, Kaiser JA, Park H-S, Liu X, Yang L, et al. (2024) Intranasal respiratory syncytial virus vaccine attenuated by codon-pair deoptimization of seven open reading frames is genetically stable and elicits mucosal and systemic immunity and protection against challenge virus replication in hamsters. PLoS Pathog 20(5): e1012198.
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Baseline mapping of severe fever with thrombocytopenia syndrome virology, epidemiology and vaccine research and development

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By Lisa Reece, Jaclyn A Kaiser, Virginia Benassi, Nathen E. Bopp
| Nature Verified
Abstract Severe fever with thrombocytopenia syndrome virus (SFTSV) is a newly emergent tick-borne bunyavirus first discovered in 2009 in China. SFTSV is a growing public health problem that may become more prominent owing to multiple competent tick-vectors and the expansion of human populations in areas where the vectors are found. Although tick-vectors of SFTSV are found in a wide geographic area, SFTS cases have only been reported from China, South Korea, Vietnam, and Japan.
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Japanese encephalitis vaccine-specific envelope protein E138K mutation does not attenuate virulence of West Nile virus

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By Jaclyn A Kaiser, Huanle Luo, Steven Widen, Thomas Wood
| Nature Verified
Abstract West Nile (WNV) and Japanese encephalitis viruses (JEV) are closely related, mosquito-borne neurotropic flaviviruses. Although there are no licensed human vaccines for WNV, JEV has multiple human vaccines, including the live, attenuated vaccine SA14-14-2. Investigations into determinants of attenuation of JE SA14-14-2 demonstrated that envelope (E) protein mutation E138K was crucial to the attenuation of mouse virulence.
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