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James Lyon

As seen in: Nature, Foreign Policy Magazine, Science Magazine, BioRxiv, Cell Metabolism, Financial Content, Asia Times, Minyanville, RealClear Defense, Samoa Observer and
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Articles

OPINION: We must associate the IDF with the evil that we know

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By James Lyon
| The Student Life Verified
(PJ James • The Student Life) From a young age, clear examples of individuals whose behavior we should not mimic or tolerate were present both in the classroom and at home — whether that be litterers, people who use their hands instead of their words or people who don’t follow the golden rule. There is a moral framework built into the way our society socializes and conditions us, especially our children.
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TBL1X/TBL1XR1 govern β-cell identity through a PAX6-containing gene regulatory network

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By Alina Walth-Hummel, Celine Jouffe, Peter Weber, Karsten Motzler, Julia Geppert, Michael Sterr, Wei Gan, Iwona Szczerbinska, Ann-Christine König, Stefanie Hauck, Daniela Hass, Congcong Wang, Kenneth A. Dyar, James Lyon, Heiko Lickert, Carina Ämmälä, Stephan Herzig, Frances M. Ashcroft, Mostafa Bakhti, Patrick Macdonald, Maria Rohm, Raul Terron-Exposito
| Nature Verified
Abstract A main mechanism of β-cell dysfunction in diabetes is loss of identity, controlled by transcription factors that induce identity gene expression and disallowed gene repression. How transcription factors facilitate simultaneous expression and repression is not fully understood, representing a knowledge gap in diabetes research.
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Persistent organic pollutant concentrations in human pancreas correlate with markers of beta cell dysfunction

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By Myriam P Hoyeck, Jana Palaniyandi, Jelena Kolic, Jillian Ashley-Martin, James Lyon, Nancy Smith, Erin E. Mulvihill, James Johnson, Patrick Macdonald, Jennifer E. Bruin, Evgenia Fadzeyeva
| Nature Verified
Abstract Epidemiological studies consistently report associations between circulating concentrations of persistent organic pollutants and increased type 2 diabetes risk. Measures of pollutant concentrations in pancreas are limited; given the role of the endocrine pancreas in diabetes pathogenesis, this is an important gap in the literature. Additionally, no studies have correlated pollutant concentrations with direct measures of human beta cell function.
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