Jean-Claude Baron
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Fifty Years of Deciphering Stroke Pathophysiology
Get full access to this article View all available purchase options and get full access to this article. References 1. Ames A, Wright RL, Kowada M, Thurston JM, Majno G. Cerebral ischemia. II. The no-reflow phenomenon. Am J Pathol. 1968;52:437–453. 2. Hossmann KA, Olsson Y. Suppression and recovery of neuronal function in transient cerebral ischemia. Brain Res. 1970;22:313–325. doi: 10.1016/0006-8993(70)90474-9 3. Hossmann KA, Sato K. Recovery of neuronal function after prolonged cerebral ischemia. Science.
Static autoregulation in humans
Get full access to this article View all access and purchase options for this article. References 1. Wang Y, Payne SJ. Static autoregulation in humans. J Cereb Blood Flow Metab 2023; 271678X231210430. Nov 7; Online ahead of print. Available from: https://pubmed.ncbi.nlm.nih.gov/37933742/ 2. Lassen NA, Agnoli A. The upper limit of autoregulation of cerebral blood flow – on the pathogenesis of hypertensive encepholopathy. Scand J Clin Lab Invest 1972; 30: 113–116. 3. Paulson OB, Strandgaard S, Edvinsson L.
Comorbidity and outcomes among hospitalized patients with stroke: a nationwide inpatient analysis
1. Introduction Comorbidities, in the context of medical research, refer to the simultaneous presence of two or more diseases in a patient, particularly in relation to an index disease under investigation (1). As the global population experiences growth and aging, coupled with elevated risk factors for non-communicable diseases, there has been an anticipated rise in the prevalence of comorbidities (2, 3).
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