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Jiabao You

As seen in: Nature, Journal of The American Academy of Dermatology (JAAD)
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Articles by Jiabao You

Targeting an IL-17/IL-36 pathogenic hub in hidradenitis suppurativa with a bispecific antibody

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By Yonghu Sun, Zihao Mi, Yueqian Yu, Zhenzhen Wang, Lele Sun, Jianke Li, Baoqi Yang, Xiuhuan Wang, Yilei Wu, Kexin Liu, Xiaochen Ren, Jing J. Liu, Hong Luan, Guowei Zhao, Jian Shi, Chi Ma, Yongxia Liu, Mingkai Zhang, Peng Song, Peng Qu, Jianfeng Wang, Na Wang, Yue Xin, Qingqing Guo, Weizhi Wu, Hongqing Tian, Xuehui Li, Guoyuan Peng, Xuan Guo, Guangliang Zhang, Benke Li, Xiaoxiao Yan, Jianwen Wang, Jiabao You, Gongqi Yu, Chuan Wang, Xiaopei Cui, Yifan Zhan, Xiangyang Zhu, Hong liu, Furen Zhang, Minkuo Zhang, Wenwen Sai, Xiuqiang Ma
| Nature Verified
Abstract Hidradenitis suppurativa (HS) is a chronic inflammatory skin disorder with a high unmet need, particularly for treatment-resistant tunnels. Integrated multiomics analysis revealed that HS tunnels are spatially organized, self-amplifying inflammatory circuits driven by synergistic IL-17/IL-36γ activation of a unique keratinocyte subset, which activates neutrophils and inflammatory fibroblasts.
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Dynamic cytokine profiles combined with enzyme-linked immunospot assay are useful for immunologically confirming the dapsone hypersensitivity syndrome

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By Jiabao You, Lele Sun, Qing Zhao, Huimin Zhang
| Journal of The American Academy of Dermatology (JAAD) Verified
To the Editor: Dapsone hypersensitivity syndrome (DHS) is a dapsone-induced T–cell mediated delayed type of hypersensitivity, characterized by fever, rash, lymphadenopathy, and hepatic function abnormalities. 1 Richardus J.H. Smith T.C. Increased incidence in leprosy of hypersensitivity reactions to dapsone after introduction of multidrug therapy. Lepr Rev. 1989; 60: 267-273 The diagnosis of DHS is traditionally based on the criteria proposed by Richardus and Smith.
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