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Low-intensity stimulation drives macrophage efferocytosis via ACSL4 lipid remodeling and CCL9-CCR1 signaling for tendon-bone healing
Abstract INTRODUCTION RESULTS DISCUSSION MATERIALS AND METHODS Acknowledgments Supplementary Materials REFERENCES Information & Authors Metrics & Citations View Options References Figures Tables Media Share Abstract The mechanisms underlying exercise rehabilitation–induced tendon-bone healing remain unclear.
IJMS | Free Full-Text | MicroRNA-124-3p Attenuated Retinal Neovascularization in Oxygen-Induced Retinopathy Mice by Inhibiting the Dysfunction of Retinal Neuroglial Cells through STAT3 Pathway
1. Introduction Retinal neovascularization is one of the primary causes of blindness in various ocular vascular diseases, including retinopathy of prematurity (ROP), age-related macular degeneration, and proliferative diabetic retinopathy. For example, ROP is one of the main causes of infantile blindness. Approximately 65.8% of infants weighing less than 1251 g and 81.6% of infants weighing less than 1000 g at birth develop ROP [1].
Fargesin ameliorates osteoarthritis via macrophage reprogramming by downregulating MAPK and NF-κB pathways
Research article Open Access Published: 14 May 2021 Jiansen Lu1,2,3,4,5 na1, Hongbo Zhang1,2,3,4 na1, Jianying Pan1,2,3,4 na1, Zhiqiang Hu1,5, Liangliang Liu1,2,3,4, Yanli Liu3,4, Xiao Yu5, Xiaochun Bai1,2,3,4, Daozhang Cai1,2,3,4 & Haiyan Zhang1,2,3,4 Arthritis Research & Therapy volume 23, Article number: 142 (2021) Cite this article Abstract Background To investigate the role and regulatory mechanisms of fargesin, one of the main components of Magnolia fargesii, in macrophage reprogramming...
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