Jiaying Du
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VMP1 restricts STING trafficking and activation through controlling CDIPT recruitment to ER membrane curvature
Abstract The cGAS-STING signaling serves as a central hub for innate immunity by orchestrating type I interferon production and inflammatory cascades in response to cytosolic DNA. A critical rate-limiting step in this pathway involves STING exit from the endoplasmic reticulum (ER), a process initiated by COPII-mediated trafficking that requires ER membrane curvature formation. However, the molecular mechanism that drives this process is poorly understood.
Correction: Case Report: Bloodstream infection due to Clostridium innocuum combined with Eggerthella lenta
A Correction on Case Report: Bloodstream infection due to Clostridium innocuum combined with Eggerthella lenta by Xu, Y., Xu, A., Du, J., Zhao, Y., Liu, X., Sun, Z., Xu, Q., Zhang, M., and Tian, Y. (2026). Front. Med. 13:1747742. doi: 10.3389/fmed.2026.1747742 Authors Anan Xu and Jiaying Du were erroneously omitted as equal contributing authors.
Case Report: Bloodstream infection due to Clostridium innocuum combined with Eggerthella lenta
CASE REPORT article Volume 13 - 2026 | https://doi.org/10.3389/fmed.2026.1747742 Background Bloodstream infection (BSI) refers to systemic inflammatory response syndrome caused by the invasion of pathogens and their toxins into the blood circulation. Blood culture, both aerobic and anaerobic, is the gold standard for diagnosing bloodstream infections (1).
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