Jin-Tae Hong on Muck Rack

Jin-Tae Hong

As seen in: Nature, MDPI
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Articles

Author Correction: Mutated IL-32θ (A94V) inhibits COX2, GM-CSF and CYP1A1 through AhR/ARNT and MAPKs/NF-κB/AP-1 in keratinocytes exposed to PM10 - Scientific Reports

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By Jinju Kim, Chae-Min Lim, Nahyun Kim, Hong-Gyum Kim, Jin-Tae Hong
| Nature Verified
Correction to: Scientific Reports https://doi.org/10.1038/s41598-024-83159-z, published online 15 January 2025 The Funding section in the original version of this Article was omitted. It now reads: “This research was supported by the National Research Foundation of Korea (NRF), South Korea (NRF-2021R1A2C3003414, NRF-2021R1A6A1A03038890, and RS-2024-00451912).” The original Article has been corrected.
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Mutated IL-32θ (A94V) inhibits COX2, GM-CSF and CYP1A1 through AhR/ARNT and MAPKs/NF-κB/AP-1 in keratinocytes exposed to PM10 - Scientific Reports

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By Jinju Kim, Nahyun Kim, Hong-Gyum Kim, Jin-Tae Hong, Chae-Min Lim
| Nature Verified
Abstract Exposure to particulate matter (PM) in the air harms human health. Most studies on particulate matter’s (PM) effects have primarily focused on respiratory and cardiovascular diseases. Recently, IL-32θ, one of the IL-32 isoforms, has been demonstrated to modulate cancer development and inflammatory responses. This study revealed that one-point mutated IL-32θ (A94V) plays an important role in attenuating skin inflammation.
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Side-Chain Immune Oxysterols Induce Neuroinflammation by Activating Microglia

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By Yonghae Son, In-Jun Yeo, Jin-Tae Hong, Seong-Kug Eo
| MDPI
1. Introduction Recent studies shed light on the intricate relationship between high cholesterol levels and Alzheimer’s disease (AD) [1,2,3,4]. Cholesterol, being highly susceptible to oxidation, undergoes enzymatic oxidation or autoxidation when exposed to oxidizing agents, resulting in the formation of oxysterols [5]. The brain, being the most cholesterol-rich organ, exhibits distinct oxysterol profiles in AD patients compared to healthy brains [2].
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