Kameljit K. Kalsi on Muck Rack

Kameljit K. Kalsi

As seen in: Nature, PLOS ONE, Physiological Reports
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Articles

Lipoxin receptor agonist and inhibition of LTA4 hydrolase prevent tight junction disruption caused by P. aeruginosa filtrate in airway epithelial cells

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By Kameljit K. Kalsi, Sonya Jackson, Deborah L. Baines
| PLOS ONE
Loading metrics Open Access Peer-reviewed Research Article Abstract Airway diseases can disrupt tight junction proteins, compromising the epithelial barrier and making it more permeable to pathogens. In people with pulmonary disease who are prone to infection with Pseudomonas aeruginosa, pro-inflammatory leukotrienes are increased and anti-inflammatory lipoxins are decreased. Upregulation of lipoxins is effective in counteracting inflammation and infection.
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Whole body hyperthermia, but not skin hyperthermia, accelerates brain and locomotor limb circulatory strain and impairs exercise capacity in humans

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By Kameljit K. Kalsi, Steven J. Trangmar, Scott T. Chiesa, Niels H. Secher
| Physiological Reports Verified
AbstractCardiovascular strain and hyperthermia are thought to be important factors limiting exercise capacity in heat?stressed humans, however, the contribution of elevations in skin (Tsk) versus whole body temperatures on exercise capacity has not been characterized.
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Hyperglycaemia and Pseudomonas aeruginosa acidify cystic fibrosis airway surface liquid by elevating epithelial monocarboxylate transporter 2 dependent lactate-H+ secretion : Scientific Reports

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By Mirko Sobotta, James Peter Garnett, Kameljit K. Kalsi, Jade Bearham
| Nature Verified
Primary human bronchial epithelial (HBE) cells (non-CF and CF) were obtained from endobronchial brushings or extracted from explanted lungs and cultured as previously described14,15.
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