Kanemasa Maki on Muck Rack

Kanemasa Maki

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Articles

Fetal Rapid Eye Movement Sleep Dysfunction as a Potential Early Indicator for NALCN‐Related CLIFAHDD Syndrome: A Case Report

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By Yoshiki Maeda, Fumiya Fukumori, Kanemasa Maki
| Wiley Online Library Verified
The sodium leak channel nonselective (NALCN) protein is a critical regulator of neuronal excitability and sleep regulation. NALCN variants, particularly gain-of-function variants, reduce rapid eye movement (REM) sleep by increasing neuronal excitability in the deep mesencephalic nucleus [1]. Fetal sleep states, including REM and non-REM sleep (NREMS), have been studied in animal models, demonstrating distinct developmental patterns during gestation.
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