Kathleen Hayden on Muck Rack

Kathleen Hayden

Verified
Washington, D.C.
As seen in: California Healthline, KFF Health News, Kaiser Family Foundation, Environmental Health Perspectives, Arthritis & Rheumatology, Nature, Wiley Online Library, Neurology, Herald Standard, Alzheimer's & Dementia, North Carolina Medical Journal, Public Health Journal, Hometown News
Covers:  health care, government policy, elections, politics,
Veteran digital journalist; editor at @KFFHealthNews and @CalHealthline; formerly @BGOV, @AOLNews, @CNN, @TIME.

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Kathleen Hayden’s Biography

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Kathleen Hayden is an online news veteran with a specialty in government, elections and policy coverage. Before joining KHN, she managed Bloomberg Government’s online, information graphics and multimedia teams and edited news and analysis on industries including health care. Prior to BGOV, Kathleen worked at AOL, CNN and TIME. She is a graduate of Haverford College and a proud New England native.

Articles

Prioritizing health and vitality for women in midlife

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By Kathleen Hayden Verified
| Hometown News Verified
As women journey through the different phases of life, their health needs evolve in unique and complex ways. Midlife, in particular, brings its own set of challenges and opportunities. As a physician with years of experience in preventive medicine and aging wellness, I believe that midlife is a pivotal time to take charge of your physical and mental well-being.
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Health and the Aging Brain

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By Kathleen Hayden Verified
| North Carolina Medical Journal
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Plasma brain-derived tau is an amyloid-associated neurodegeneration biomarker in Alzheimer’s disease - Nature Communications

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By Fernando Gonzalez-Ortiz, Jose Celso Contador, Geir Bråthen, Przemyslaw R. Kac, Johanna Nilsson, Kathleen Hayden Verified, Albert Puig-Pijoan, Henrik Zetterberg, Marc Suárez-Calvet, Thomas K. Karikari, Tormod Fladby
| Nature Verified
Abstract Staging amyloid-beta (Aβ) pathophysiology according to the intensity of neurodegeneration could identify individuals at risk for cognitive decline in Alzheimer’s disease (AD). In blood, phosphorylated tau (p-tau) associates with Aβ pathophysiology but an AD-type neurodegeneration biomarker has been lacking.
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