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Stimulation of Angiotensin II Receptor Subtype 2 Reduces Preeclampsia-like Symptoms in a Mouse Model of Preeclampsia
All articles published by MDPI are made immediately available worldwide under an open access license. No special permission is required to reuse all or part of the article published by MDPI, including figures and tables. For articles published under an open access Creative Common CC BY license, any part of the article may be reused without permission provided that the original article is clearly cited. For more information, please refer to https://www.mdpi.com/openaccess.
Human UC-MSC-derived exosomes facilitate ovarian renovation in rats with chemotherapy-induced premature ovarian insufficiency
Introduction Premature ovarian insufficiency (POI) refers to a clinical syndrome in which women younger than 40 years develop ovarian dysfunction, which is mainly characterized by abnormal menstruation (amenorrhea or oligomenorrhea for at least 4 months), increased gonadotropin levels (an elevated follicle-stimulating hormone (FSH) level > 25 IU/L on two occasions > 4 weeks apart), and oestrogen-deficiency symptom (1).
The Influence of Nicotine on Trophoblast-Derived Exosomes in a Mouse Model of Pathogenic Preeclampsia
Abstract : Preeclampsia (PE) is a serious complication of pregnancy with a pathogenesis that is not fully understood, though it involves the impaired invasion of extravillous trophoblasts (EVTs) into the decidual layer during implantation.
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