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Highlights • A limited set of mRNA transcripts requires spliceosome methylation for processing • PRMT5 has a conserved role in the induction of antiviral interferon responses • PRMT5 controls IFNB1 and IFNL1 transcription via activation of ATF2, c-Jun, and TBK1 • Altered ATF2 splicing is insufficient on its own to repress interferon signaling Summary Alternative splicing is well understood to enhance proteome diversity as cells respond to stimuli.
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