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Kenneth Barentsen

As seen in: Wiley Online Library, MDPI
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Articles

Trophoblast‐specific overexpression of Slc7a5 increases fetal amino acid availability and hepatic mTOR signalling and causes increased fetal growth

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By Fredrick J Rosario, Hiroshi Shimada, Kenneth Barentsen, Theresa Powell
| Wiley Online Library Verified
Abnormal fetal growth is linked to perinatal complications and increased risk of metabolic and cardiovascular diseases, yet the underlying mechanisms remain unclear. Placental System L amino acid transport is decreased in human pregnancies complicated by fetal growth restriction and increased in fetal overgrowth. We previously demonstrated that trophoblast-specific overexpression (OX) of Slc7a5 /LAT1 enhances transplacental leucine transport and fetal growth.
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Nutrients | Free Full-Text | Knockdown of Placental Major Facilitator Superfamily Domain Containing 2a in Pregnant Mice Reduces Fetal Brain Growth and Phospholipid Docosahexaenoic Acid Content

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By Theresa Powell, Owen Vaughan, Charis Uhlson, Kenneth Barentsen
| MDPI
All articles published by MDPI are made immediately available worldwide under an open access license. No special permission is required to reuse all or part of the article published by MDPI, including figures and tables. For articles published under an open access Creative Common CC BY license, any part of the article may be reused without permission provided that the original article is clearly cited. For more information, please refer to https://www.mdpi.com/openaccess.
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