Is this you? As a journalist, you can create a free Muck Rack account to customize your profile, list your contact preferences, and upload a portfolio of your best work.
Claim your profile
Articles by Kumar Uddipto
Hypomethylating agents induce epigenetic and transcriptional heterogeneity with implications for AML cell self-renewal.
A.K.E. declares the following competing interests: advisory board and honoraria from AbbVie, Astellas, Gilead, Servier, Jazz, Otsuka/Astex, and RACE Oncology; speaker fees from AbbVie, Otsuka/Astex, Astellas and Jazz; Research Funding from RACE oncology and Otsuka/Astex. N.A.B. receives research funding from Merck KGaA and Bristol Myers Squibb.
The Existence of an Intra-Amniotic Microbiome: Assessing a Controversy
All articles published by MDPI are made immediately available worldwide under an open access license. No special permission is required to reuse all or part of the article published by MDPI, including figures and tables. For articles published under an open access Creative Common CC BY license, any part of the article may be reused without permission provided that the original article is clearly cited. For more information, please refer to https://www.mdpi.com/openaccess.
Upregulated cholesterol biosynthesis facilitates the survival of methylation-retaining AML cells following decitabine treatment.
Abstract DNA hypomethylating agents (HMAs) are used to treat acute myeloid leukaemia (AML) and myelodysplasia patients who are unsuitable for intensive chemotherapy, but low response rates and therapy-resistant relapse remain significant challenges. To optimise HMA efficacy, we must understand how resistance and relapse arise from cells that survive treatment.
Insights into the Genital Microbiota of Women Who Experienced Fetal Death in Utero
Open AccessArticle by 1, 2, 3, 3, 1,4 and 2,* 1 College of Health and Medicine, Australian National University, Canberra, ACT 2601, Australia 2 School of Medicine, Sydney, University of Notre Dame Australia, Darlinghurst, NSW 2010, Australia 3 Área de Ciencias Biomédicas y Policlínico, University of Piura, San Eduardo, Piura 20009, Peru 4 Institute for Health Research, University of Notre Dame Australia, Fremantle, WA 6160, Australia * Author to whom correspondence should be addressed.
Single-cell epigenomics in cancer: charting a course to clinical impact
The epigenome The mammalian epigenome comprises numerous molecular mechanisms that regulate the accessibility of genomic information. Genes involved in the function of a particular cell type are maintained in an accessible state, while unrequired genes are epigenetically silenced. Thus, epigenetic mechanisms restrict the potential of each cell type and are essential for mammalian development [1].
Show More
loading
Actions
Is this you?
As a journalist, you can create a free Muck Rack account to customize your profile, list your contact preferences, and upload a portfolio of your best work.Get in touch with Kumar
Contact Kumar, search articles and posts on X, monitor coverage, and track replies from one place.
Learn more about Muck Rack