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Cancer-associated metabolic alterations facilitated by astrocyte glycogenolysis mediate neuropathic pain chronification

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By Junseo Park, Hyewon Jun, Sung Lee, Kwanghwan Kim
| biorxiv.org
Abstract Metabolic signaling in astrocytes has been implicated in a range of neurophysiological disorders. Likewise, pain chronification, which is defined as the transition from an acute pain state to a persistent one in response to repeated or intense nociceptive stimuli, involves crucial contributions from astrocytes.
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