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To the Editor, Acute myeloid leukemia (AML) is a highly aggressive hematologic malignancy characterized by myeloproliferation, a block in differentiation, and accumulation of immature blasts in the bone marrow and peripheral blood [1, 2]. The pathogenesis of AML is genetically diverse, with recurrent chromosomal and molecular alterations that drive leukemogenesis and influence prognosis and treatment decisions [1, 2].
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