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Kyoungwha Pang

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Articles

ESRP1-regulated DNMT3B isoform switching determines the malignant potential of pancreatic cancer

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By Eunji Hong, HyeYeon Park, Junil Kim, Jin Muk Kang, Sujin Park, Taehong Min, Jinah Park, Kyoungwha Pang, Pyunggang Kim, Minjung Son, Akira Ooshima, Daeun Kim, Wooil Kwon, Hideyuki Takeshima, Ja-Lok Ku, Hongbeom Kim, Toshikazu Ushijima, Jin-Young Jang, Yong Jung Kwon, Daechan Park, Seok Hee Park, Seong-Jin Kim, Jeongeun Im
| Nature Verified
Abstract Epithelial splicing regulatory protein 1 (ESRP1) is a key regulator of epithelial-mesenchymal transition (EMT) and cancer progression, including in pancreatic ductal adenocarcinoma (PDAC). We show that ESRP1-mediated isoform switching of DNA methyltransferase 3B (DNMT3B) correlates with the metastatic potential of pancreatic cancer cells. The expression of DNMT3B splicing variants was closely linked to ESRP1 level.
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Destablilization of TRAF6 by DRAK1 Suppresses Tumor Growth and Metastasis in Cervical Cancer Cells

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By Yuna Park, Seongnam City, Kyoungwha Pang, Jinah Park
| cancerres.aacrjournals.org
Molecular Cell Biology Yuna Park, Kyoungwha Pang, Jinah Park, Eunji Hong, Jihee Lee, Akira Ooshima, Hae-Suk Kim, Jae Hyun Cho, Youngjin Han, Cheol Lee, Yong Sang Song, Kyung-Soon Park, Kyung-Min Yang and Seong-Jin Kim DOI: 10.1158/0008-5472.CAN-19-3428 Published June 2020 This article requires a subscription to view the full text. You may purchase access to this article or login to access your subscription using the links below.
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Destabilization of TRAF6 by DRAK1 suppresses tumor growth and metastasis in cervical cancer cells

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By Yuna Park, Biomedical Science, Kyoungwha Pang, Jinah Park
| cancerres.aacrjournals.org
Research Article Yuna Park, Kyoungwha Pang, Jinah Park, Eunji Hong, Jihee Lee, Akira Ooshima, Hae-Suk Kim, Jae Hyun Cho, Youngjin Han, Cheol Lee, Yong Sang Song, Kyung-Soon Park, Kyung-Min Yang and Seong-Jin Kim DOI: 10.1158/0008-5472.CAN-19-3428 Abstract The adaptor protein TNF receptor-associated factor 6 (TRAF6) is a key mediator in inflammation. However, the molecular mechanisms controlling its activity and stability in cancer progression remain unclear.
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