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Microvascular outcomes of a pregnancy complicated by preeclampsia
Background Preeclampsia (PE) in pregnancy is associated with future cardiovascular disease through poorly understood mechanisms. Endothelial dysfunction persists in women following PE in pregnancy. We hypothesized that microvascular endothelial dysfunction associated with PE may be a postpartum mechanism of incipient cardiovascular disease. The purpose of this study was to examine the association of prior PE to postpartum changes in maternal microvascular endothelial function.
Measurement of concentrations of four chemical ultraviolet filters in human breast tissue at serial locations across the breast
The human population is widely exposed to benzophenone‐3 (BP‐3), octylmethoxycinnamate (OMC), 4‐methylbenzilidenecamphor (4‐MBC) and homosalate from their use in consumer goods to absorb ultraviolet (UV) light. Their oestrogenic activity and presence in human milk suggest a potential to influence breast cancer development.
Examining the Microvasculature Following a Pregnancy Complicated by Preeclampsia
To view the full text, please login as a subscribed user or purchase a subscription. Click here to view the full text on ScienceDirect. Pre-eclampsia (PE) is a maternal hypertensive disorder associated with elevated lifetime risk for cardiovascular disease. Microvascular dysfunction- imbalances in vasodilatory potential- has been reported in women following a pregnancy complicated by PE.
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