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Cover Image, Volume 239, Number 11, November 2024
Simon A. Mallal Division of Infectious Diseases, Vanderbilt University Medical Center, Nashville, Tennessee, USA Institute for Immunology and Infectious Diseases, Murdoch University, Murdoch, Western Australia, Australia Search for more papers by this author
Cytokine and chemokine receptor profiles in adipose tissue vasculature unravel endothelial cell responses in HIV
1 INTRODUCTION The vascular endothelium is highly pleiotropic and can change phenotype from an anti-inflammatory, antithrombotic, and pro-vasodilatory state to one that favors inflammation, thrombosis, and vasoconstriction. This switch in phenotype precedes the development of atherosclerotic cardiovascular disease (CVD) and is often referred to as endothelial dysfunction (Bonetti et al., 2003; Davignon & Ganz, 2004).
Inflammatory and Immune Mechanisms for Atherosclerotic Cardiovascular Disease in HIV
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